A CRF02_AGa CRF02_AGa CRF02_AGaCRF02_AG CRF22_01A1 CRF02_AG CRF02_AG CRF36_cpxb/F2b CRF02_AG NDc CRF02_AG CRF02_AG ND NDc cCRF02_AG CRF01_AE CRF02_AG CRF02_AG CRF01/F CRF02_AG A-likeb CRF02_AG CRF02_AG CRF01_AE CRF02_AG NDc CRF02_AGNDc CRF02_AG A1 A1 F G A1 CRF02_AG CRF02_AG CRF02_AG CRF02_AG CRF02_AG CRF02_AG CRF02_AG CRF02_AG CRF37_cpx F CRF01_AE CRF37_cpx Ub DCRF02_AGa CRF02_AG URF A1 URF G URF

Assays of GBM cells stably transfected to over-express galectin-1, perfectly fit in with the previous studies mentioned above and highlight the importance of galectin-1 in the biologically aggressive behavior of experimental GBMs. While there was no enhancement of proliferation or change inattachment to fibronectin, galectin-1 upregulation induced more rapid two-dimensional migration and enhanced